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Search results (163)

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Loss of REST associated with Alzheimer's disease pathology is ameliorated by NAD.

Lagartos-Donate MJ. et al, (2026), Brain

In reply to Letter to the Editor from Paudel: Comment on "Changes in iPSC-Astrocyte Morphology Reflect Alzheimer's Disease Patient Clinical Markers".

Buckley NJ. and Rowland HA., (2025), Stem Cells, 43

Changes in iPSC-astrocyte morphology reflect Alzheimer's disease patient clinical markers.

Rowland HA. et al, (2025), Stem Cells, 43

Multiomics profiling of human plasma and cerebrospinal fluid reveals ATN-derived networks and highlights causal links in Alzheimer's disease.

Shi L. et al, (2023), Alzheimers Dement, 19, 3350 - 3364

Deep learning-based cell profiling based on neuronal morphology

Liu Q. et al, (2023)

Temporal transcriptional control of neural induction in human induced pluripotent stem cells.

Gupta S. et al, (2023), Front Mol Neurosci, 16

Multiomics machine learning identifies inflammation molecular pathways in prodromal Alzheimer’s Disease

Gómez-Pascual A. et al, (2023)

Glycosylated clusterin species facilitate Aβ toxicity in human neurons.

Foster EM. et al, (2022), Sci Rep, 12

Repressor element 1-silencing transcription factor deficiency yields profound hearing loss through Kv7.4 channel upsurge in auditory neurons and hair cells.

Zhang H. et al, (2022), Elife, 11

Glycosylated clusterin species facilitate Aβ toxicity in human neurons

Foster EM. et al, (2022)

Predicting AT(N) pathologies in Alzheimer's disease from blood-based proteomic data using neural networks.

Zhang Y. et al, (2022), Front Aging Neurosci, 14

Neurons derived from individual early Alzheimer's disease patients reflect their clinical vulnerability.

Ng B. et al, (2022), Brain Commun, 4

Multiomics profiling of human plasma and CSF reveals ATN derived networks and highlights causal links in Alzheimer’s disease

Shi L. et al, (2022)

Repressor element 1-silencing transcription factor deficiency yields profound hearing loss through K v 7.4 channel upsurge in auditory neurons and hair cells

Zhang H. et al, (2022)

Neurons from individual early Alzheimer’s disease patients reflect their clinical vulnerability

Ng B. et al, (2021)

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