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Search results (12)

« Back to Publications

iPSC-Astrocyte morphology reflects patient clinical markers

Rowland HA. et al, (2023)

Neurons derived from individual early Alzheimer's disease patients reflect their clinical vulnerability.

Ng B. et al, (2022), Brain Commun, 4

Neurons from individual early Alzheimer’s disease patients reflect their clinical vulnerability

Ng B. et al, (2021)

Increased Axon Initial Segment Length Results in Increased Na+ Currents in Spinal Motoneurones at Symptom Onset in the G127X SOD1 Mouse Model of Amyotrophic Lateral Sclerosis.

Jørgensen HS. et al, (2021), Neuroscience, 468, 247 - 264

Pro-maturational Effects of Human iPSC-Derived Cortical Astrocytes upon iPSC-Derived Cortical Neurons.

Hedegaard A. et al, (2020), Stem Cell Reports, 15, 38 - 51

Shorter axon initial segments do not cause repetitive firing impairments in the adult presymptomatic G127X SOD-1 Amyotrophic Lateral Sclerosis mouse.

Bonnevie VS. et al, (2020), Sci Rep, 10

Honing the Double-Edged Sword: Improving Human iPSC-Microglia Models.

Hedegaard A. et al, (2020), Front Immunol, 11

Increased axon initial segment length results in increased Na+currents in spinal motoneurones at symptom onset in the G127X SOD1 mouse model of Amyotrophic Lateral Sclerosis

Jørgensen HS. et al, (2020)

The Subprimary Range of Firing Is Present in Both Cat and Mouse Spinal Motoneurons and Its Relationship to Force Development Is Similar for the Two Species.

Jensen DB. et al, (2018), J Neurosci, 38, 9741 - 9753

Potassium channel abnormalities are consistent with early axon degeneration of motor axons in the G127X SOD1 mouse model of amyotrophic lateral sclerosis.

Maglemose R. et al, (2017), Exp Neurol, 292, 154 - 167

ctivity-Dependent Exocytosis of Lysosomes Regulates the Structural Plasticity of Dendritic Spines.

Padamsey Z. et al, (2017), Neuron, 93, 132 - 146

Postactivation depression of the Ia EPSP in motoneurons is reduced in both the G127X SOD1 model of amyotrophic lateral sclerosis and in aged mice.

Hedegaard A. et al, (2015), J Neurophysiol, 114, 1196 - 1210