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The environment and events that we are exposed to in utero, during birth and in early childhood influence our future physical and mental health. The underlying mechanisms that lead to these outcomes are unclear, but long-term changes in epigenetic marks, such as DNA methylation, could act as a mediating factor or biomarker. DNA methylation data were assayed at 713 522 CpG sites from 9537 participants of the Generation Scotland: Scottish Family Health Study, a family-based cohort with extensive genetic, medical, family history and lifestyle information. Methylome-wide association studies of eight early life environment phenotypes and two adult mental health phenotypes (major depressive disorder and brief resilience scale) were conducted using DNA methylation data collected from adult whole blood samples. Two genes involved with different developmental pathways (PRICKLE2, Prickle Planar Cell Polarity Protein 2 and ABI1, Abl-Interactor-1) were annotated to CpG sites associated with preterm birth (P 

Original publication

DOI

10.1093/hmg/ddab274

Type

Journal article

Journal

Hum Mol Genet

Publication Date

21/02/2022

Volume

31

Pages

651 - 664

Keywords

Adaptor Proteins, Signal Transducing, Child, Preschool, CpG Islands, Cytoskeletal Proteins, DNA Methylation, Depressive Disorder, Major, Epigenesis, Genetic, Epigenome, Female, Humans, Infant, Newborn, Mental Health, Pregnancy, Premature Birth