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Airway inflammation is central to the pathogenesis of allergic asthma, and molecules that mediate this process obviously represent targets for therapy. In the present article, we discuss our experiments, which point to CD4+ T cells and IL-5-driven eosinophilia as potential targets for the relief of bronchial hyperreactivity in late-phase asthma.

Original publication

DOI

10.1046/j.1440-1711.1998.00766.x

Type

Journal article

Journal

Immunol Cell Biol

Publication Date

10/1998

Volume

76

Pages

454 - 460

Keywords

Animals, Asthma, CD4-Positive T-Lymphocytes, Cell Movement, Chemokine CCL11, Chemokines, CC, Cytokines, Disease Models, Animal, Eosinophils, Immunity, Cellular, Interleukin-4, Interleukin-5, Lung, Mice, Pulmonary Eosinophilia